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In neurodegenerative disease models, the co-delivery of GLP-1 analogues and conformation-specific antibodies against A β or α-syn has achieved concurrent neuroprotection and proteopathy clearance, leading to sustained behavioral and histological improvements [437, 438]
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D-Nle-Tyr-Ile-NH-(CH 2 ) 5 -CONH 2 also contributing to its slow removal from the blood was a lack of Phase I metabolism for D-Nle-Tyr-Ile-NH-(CH 2 ) 5 -CONH 2
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