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Key Takeaways L-cysteine is the rate-limiting amino acid for glutathione synthesis — without adequate cysteine supply, your dog’s most important antioxidant defence system cannot function optimally.¹˒² Clinically ill dogs demonstrate significantly lower glutathione concentrations than healthy dogs, and this depletion correlates with both disease severity and poorer outcomes.¹ Cysteine-based supplementation in hospitalised dogs has been shown to increase plasma cysteine and maintain glutathione concentrations that otherwise decline during illness.³ Cysteine supports liver detoxification through the trans-sulphuration pathway, providing the sulphur substrate required for hepatic phase II conjugation reactions.⁴ Research in intestinal models demonstrates that cysteine enhances gut barrier function by reducing oxidative stress-induced tight junction permeability and suppressing pro-inflammatory cytokine expression.⁵˒⁶ L-cysteine in Boost works alongside DL-methionine, taurine, milk thistle and vitamin C to deliver multi-pathway antioxidant and hepatoprotective support

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BAP1 can reduce the H2Aub occupancy on the promoter of SLC7A11 in a deubiquitinating dependent manner, inhibit the expression of SLC7A11 and decrease the uptake of cystine, thus leading to lipid peroxidation and ferroptosis