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The signaling pathway associated with ferroptosis is mainly split into two main pathways: The exogenous, transporter-dependent pathway, which is derived from the amino acid anti-porter system X c − inhibition of iron carrier activation, and the intrinsic, enzyme-dependent pathway, which is derived from the reduction of antioxidant enzymes such as GSH or GPX4 [48,49,50]

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Most of these compounds, however, have low metabolic stability, which is not ideal for radioligand therapy,” noted Thomas Günther, PhD, pharmaceutical radiochemist at Stanford University in Stanford, California