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LPS could activate airway epithelial cells, neutrophils, and alveolar macrophages, resulting in the release of numerous inflammatory mediators, such as reactive oxygen species (ROS), tumor necrosis factor-α (TNF-α), and interleukin-6 (IL-6)

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Since epithelial–mesenchymal interactions are not restricted to embryonic development, they have been highlighted as potential targets for future therapies

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